Books about Estrogenic from Amazon.com

• Lower your risk of metabolic disorders, disease, and chronic weight gain
• Protect yourself against the estrogenic substances in the environment, products, water, and food

Learn how certain foods and herbs can protect you!

Estrogenic chemicals—known for causing the near extinction of various living species—are found in some of the most common foods we eat.

In this revolutionary diet book, Ori Hofmekler addresses the millions of overweight and obese individuals who have failed or are disappointed with other diets—those who suffer from yoyo dieting, weight gain rebounds, or accumulation of stubborn fat in the belly and other estrogen-sensitive areas. Focusing on our current over-exposure to estrogenic chemicals in the environment, foods, and water, The Anti-Estrogenic Diet provides a practical solution to fat gain, estrogen-related disorders (PMS, endometriosis, fibrocystic disease), and increased risk of common cancers in women and men (breast, ovarian, cervical, prostate).

Allowing you to still enjoy your favorite foods, the program is based on incorporating anti-estrogenic foods, spices, and herbs into your diet, while eliminating estrogenic foods and chemicals. Exposing dietary myths and fallacies, Hofmekler teaches readers that some foods commonly regarded as “healthy” may actually be harmful and vice versa.

Special chapters dedicated to readers with different needs and health conditions, recipes, a question-and-answer section, and a list of scientific references are also included in this valuable resource..
Price: $7.35 [Notify me when price goes down.]

This digital document is a journal article from Environmental Pollution, published by Elsevier in 2007. The article is delivered in HTML format and is available in your Amazon.com Media Library immediately after purchase. You can view it with any web browser.

Description:
Estrogenic potencies of the effluents or water samples from wastewater treatment plants (WWTPs), industries and hospitals and some receiving rivers in Beijing city were estimated by using a human estrogen receptor recombinant yeast assay. Estrogenic activity of industrial wastewaters was found to range from 0.1 to 13.3ng EEQ/L and decreased to the range of 0.03-1.6ng EEQ/L after treatment. Estrogenic activity in WWTP influent ranged from 0.3 to 1.7ng EEQ/L and decreased to the range of 0.05-0.5ng EEQ/L after treatment. In the receiving river waters, the estrogenic effect range was 0.1-4.7ng EEQ/L. These data suggest that treated industrial effluents and WWTP effluents of concern are not the only source of estrogenic pollution in surface waters in Beijing city. EEQ levels in Beijing river water are likely attributable to untreated municipal and industrial wastewaters discharged directly into the river. .
Price: $10.95 [Notify me when price goes down.]

This digital document is an article from Environmental Health Perspectives, published by Thomson Gale on November 1, 2006. The length of the article is 2046 words. The page length shown above is based on a typical 300-word page. The article is delivered in HTML format and is available in your Amazon.com Digital Locker immediately after purchase. You can view it with any web browser.

Citation Details
Title: The feed factor: estrogenic variability in lab animal diets.(NIEHS News)
Author: M. Nathaniel Mead
Publication:Environmental Health Perspectives (Magazine/Journal)
Date: November 1, 2006
Publisher: Thomson Gale
Volume: 114 Issue: 11 Page: A640(3)

Distributed by Thomson Gale.
Price: $9.95 [Notify me when price goes down.]

This digital document is an article from Environmental Health Perspectives, published by National Institute of Environmental Health Sciences on March 1, 2005. The length of the article is 7696 words. The page length shown above is based on a typical 300-word page. The article is delivered in HTML format and is available in your Amazon.com Digital Locker immediately after purchase. You can view it with any web browser.

Citation Details
Title: Binding of estrogenic compounds to recombinant estrogen receptor-[alpha]: application to environmental analysis.(Research)
Author: Arnaud Pillon
Publication:Environmental Health Perspectives (Refereed)
Date: March 1, 2005
Publisher: National Institute of Environmental Health Sciences
Volume: 113 Issue: 3 Page: 278(7)

Distributed by Thomson Gale.
Price: $5.95 [Notify me when price goes down.]

This digital document is an article from Environmental Health Perspectives, published by Thomson Gale on January 1, 2005. The length of the article is 5587 words. The page length shown above is based on a typical 300-word page. The article is delivered in HTML format and is available in your Amazon.com Digital Locker immediately after purchase. You can view it with any web browser.

Citation Details
Title: Human colon microbiota transform polycyclic aromatic hydrocarbons to estrogenic metabolites.(Research)
Author: Tom Van de Wiele
Publication:Environmental Health Perspectives (Magazine/Journal)
Date: January 1, 2005
Publisher: Thomson Gale
Volume: 113 Issue: 1 Page: 6(5)

Distributed by Thomson Gale.
Price: $5.95 [Notify me when price goes down.]

This digital document is a journal article from Chemosphere, published by Elsevier in 2006. The article is delivered in HTML format and is available in your Amazon.com Media Library immediately after purchase. You can view it with any web browser.

Description:
This work evaluated the joint anti-estrogenic effects of pentachlorophenol (PCP) and 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) against 17@b-estradiol (E2) in juvenile goldfish (Carassius auratus) hepatocyte cultures. The level of vitellogenin (VTG) as a biomarker was determined by exposing hepatocytes to individual E2, PCP and TCDD, as well as to E2 in the presence of PCP, TCDD or their mixtures of various concentrations. PCP and TCDD did not exhibit estrogenicity. Both chemicals reduced the estrogenicity of E2, indicating the anti-estrogenic effects of PCP and TCDD. Their anti-estrogenic EC"5"0 values were calculated. The joint anti-estrogenic effects against E2 increased with increasing the PCP-to-TCDD ratio of mixture. Marking's indices were <0, suggesting an antagonism in anti-estrogenic effects between PCP and TCDD. The anti-estrogenic effects of PCP appeared to result primarily from the competitive binding to estrogen receptor. While TCDD may undergo an indirect binding process for its anti-estrogenic effects, the accurate mechanisms remain to be understood. The observed antagonism in anti-estrogenic effects resulted apparently from the mutual inhibition by PCP and TCDD. .
Price: $10.95 [Notify me when price goes down.]

This digital document is a journal article from Science of the Total Environment, The, published by Elsevier in 2007. The article is delivered in HTML format and is available in your Amazon.com Media Library immediately after purchase. You can view it with any web browser.

Description:
A suite of 30 primarily estrogenic organic wastewater contaminants was measured in several influent/effluent wastewater samples from four municipal wastewater treatment plants and effluents from one bleached kraft pulp mill (BKME) using an ultra-trace analytical method based on gas chromatography-high resolution mass spectroscopy (GC-HRMS). In vitro recombinant yeast assay detection of the estrogenic equivalent (EEq) on whole and solid phase extracted (SPE) and fractionated wastewater was also performed. 19-Norethindrone was the most frequently detected and abundant (26-224 ng/L) of all the synthetic estrogens/progesterones in the influent samples. 17@a-Ethinylestradiol was the more frequently detected synthetic estrogen/progesterone in the effluents occurring at or below 5 ng/L with some sporadic occurrences of up to 178 ng/L. The greatest levels of steroidal estrogens in municipal effluents were E1>E2>E3 which were all <20 ng/L. Nonylphenol and di(2-ethylhexyl) phthalate were found to be the highest non-steroidal synthetic compounds surveyed in both municipal influent and effluent samples, both occurring at 6-7 @mg/L in municipal effluents. BKME contained relatively large amounts of the plant sterol stigmasterol (4 @mg/L) but low amounts of fecal sterols, and steroidal estrogens (E2 only at 6 ng/L) when compared to the municipal effluents. In vitro EEq in the wastewater surveyed ranged from 9-106 ng E2/L and ranked from municipal influent>municipal effluent~BKME, with most of the estrogenicity fractionating in the 100% methanol SPE fraction followed by a secondary amount in the diethyl ether (for municipal) or methyl-tert butyl ether (for BKME) SPE fractions. Most correlations between chemical and in vitro estrogenic equivalency were weak (p>0.05 in most cases). Unexpected inverse correlations between in vitro estrogenic activity and concentrations of the estrogenic contaminant bisphenol A were found which likely contributed to the weakness of these correlations. A modified toxicity identification and evaluation procedure was continued with the SPE extracts from the more potent 100% methanol SPE fractions of municipal effluent. High performance liquid chromatography band elution retention times, based on in vitro estrogen detection, indicated that steroidal estrogens such as E2 were responsible for most of the estrogenicity of the samples. Subsequent collection and GC-MS analysis of active bands did not confirm the presence of steroidal estrogens, but expanded the possibility of phthalate esters (i.e. dibutyl phthalate) and natural sterols (i.e. @b-sitosterol) contributing to the overall estrogenic load. .
Price: $10.95 [Notify me when price goes down.]

This digital document is a journal article from Science of the Total Environment, The, published by Elsevier in 2006. The article is delivered in HTML format and is available in your Amazon.com Media Library immediately after purchase. You can view it with any web browser.

Description:
Endocrine disrupting effects on fish associated with sewage treatment effluents have been demonstrated in several studies. To investigate if the effluents from two modern Swedish sewage treatment plants contained estrogenic substances, juvenile rainbow trout were exposed to sewage water before and after the last treatment step which is a sand filter. As a biomarker for estrogenic effect, vitellogenin was analysed in the blood plasma of the exposed fish. To identify substances possibly responsible for the effect, bile fluid from the exposed fish were analysed with GC/MS. Elevated levels of vitellogenin were only seen in the fish exposed at one of the sewage treatment plants, the one with shorter residence time in the biological treatment steps, which suggests that the residence time is of importance for the ability to reduce the amount of estrogenic substances in the sewage water. The highest elevation of vitellogenin was seen in the fish exposed to water before the sand filter, which indicates that the sand filter contributes to further reduction of estrogenic substances in the sewage water. In bile from the same group of fish, considerably higher concentrations of estrone, bisphenol A and 4-nonylphenol (4.0 @mg/g bile, 23 @mg/g bile and 24 @mg/g bile, respectively) were observed compared to bile from control fish (<0.04 @mg/g bile, 0.21 @mg/g bile, and 3.5 @mg/g bile, respectively). The more potent steroidal estrogens were suggested to be major contributors to the observed estrogenic effect, although xenoestrogens were detected at higher concentrations in the bile fluid. .
Price: $10.95 [Notify me when price goes down.]

This digital document is an article from Environmental Health Perspectives, published by Thomson Gale on January 1, 2006. The length of the article is 6388 words. The page length shown above is based on a typical 300-word page. The article is delivered in HTML format and is available in your Amazon.com Digital Locker immediately after purchase. You can view it with any web browser.

Citation Details
Title: The estrogenic effect of bisphenol a disrupts pancreatic [beta]-cell function in vivo and induces insulin resistance.(Research)
Author: Paloma Alonso-Magdalena
Publication:Environmental Health Perspectives (Magazine/Journal)
Date: January 1, 2006
Publisher: Thomson Gale
Volume: 114 Issue: 1 Page: 106(7)

Distributed by Thomson Gale.
Price: $5.95 [Notify me when price goes down.]